Article
April 24, 2017
Gladstone scientists identify inflammatory pathways as a potential therapeutic target for frontotemporal dementia
Gan Lab
Aging
Immunology
Article
April 11, 2017
Gladstone and UCSF scientists discover shared characteristic among neurodegenerative diseases that occur across the lifespan.
Gan Lab
Article
March 30, 2017
Li Gan's innovative research and admirable mentorship led to her appointment as associate director of the Gladstone Institute of Neurological Disease.
Gan Lab
Article
January 11, 2017
Scientists from the Gladstone Institutes will team up with other national experts to better understand frontotemporal dementia.
Gan Lab
Article
August 30, 2016
CRISPR has enormous potential to cure intractable diseases. At the Gladstone Institutes, scientists are using the technology to advance scientific knowledge and pursue new therapies for heart disease, HIV, dementia, blindness, and more.
Conklin Lab
Ding Lab
Gan Lab
CRISPR/Gene Editing
Article
June 9, 2016
Scientists at the Gladstone Institutes take a comprehensive approach to dementia research, pursuing several potential causal factors of the disease in the hopes of discovering breakthrough treatments.
Alzheimer’s Disease
Gan Lab
Huang Lab
Mucke Lab
Article
March 30, 2016
Gladstone scientists have discovered how tau disrupts the ability of brain cells to form new memories in Alzheimer’s disease.
Alzheimer’s Disease
Gan Lab
Article
September 21, 2015
By repurposing a prescription drug used to treat rheumatoid arthritis, researchers at the Gladstone Institutes successfully reversed tau-related symptoms in an animal model of dementia.
Alzheimer’s Disease
Gan Lab
Article
August 24, 2014
SAN FRANCISCO, CA—Researchers at the Gladstone Institutes and University of California, San Francisco have shown that a loss of cells in the retina is one of the earliest signs of frontotemporal dementia (FTD) in people with a genetic risk for the disorder—even before any changes appear in their behavior.
Neurological Disease
Gan Lab
Article
December 2, 2009
SAN FRANCISCO, CA—Stimulating the growth of new neurons to replace those lost in Alzheimer's disease (AD) is an intriguing therapeutic possibility. But will the factors that cause AD allow the new neurons to thrive and function normally?
Alzheimer’s Disease
Gan Lab
Huang Lab