July 14, 2014
SAN FRANCISCO, CA—A new study from the Gladstone Institutes has revealed a way to alleviate the learning and memory deficits caused by apoE4, the most important genetic risk factor for Alzheimer’s disease, improving cognition to normal levels in aged mice.
Huang Lab
Alzheimer’s Disease
August 28, 2013
SAN FRANCISCO, CA—There is no easy way to study diseases of the brain. Extracting brain cells, or neurons, from a living patient is difficult and risky, while examining a patient’s brain post-mortem usually only reveals the disease’s final stages.
Huang Lab
Alzheimer’s Disease
Stem Cells/iPSCs
December 4, 2012
SAN FRANCISCO, CA—Scientists at the Gladstone Institutes are recommending a shift in efforts to treat Alzheimer’s disease by developing drugs that target the apoE4 protein, which has long been associated with an increased risk for the disease.
Alzheimer’s Disease
Huang Lab
Mahley Lab
Article
October 12, 2010
SAN FRANCISCO, CA—Alzheimer's disease is an extremely complicated disease. Several proteins seem to be involved in its cause and progression.
Alzheimer’s Disease
Huang Lab
Article
December 2, 2009
SAN FRANCISCO, CA—Stimulating the growth of new neurons to replace those lost in Alzheimer's disease (AD) is an intriguing therapeutic possibility. But will the factors that cause AD allow the new neurons to thrive and function normally?
Alzheimer’s Disease
Gan Lab
Huang Lab
