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Recent Advances

April 26, 2012

SAN FRANCISCO, CA—April 26, 2012—Scientists at the Gladstone Institutes have unraveled a process by which depletion of a specific protein in the brain contributes to the memory problems associated with Alzheimer's disease.

March 28, 2012

SAN FRANCISCO, CA—Scientists at the Gladstone Institutes have discovered a key protein that regulates insulin resistance—the diminished ability of cells to respond to the action of insulin and which sets the stage for the development of the most common form of diabetes.

January 25, 2012

SAN FRANCISCO, CA—Scientists at the Gladstone Institutes, an independent and nonprofit biomedical-research organization, have identified a protein that exacerbates symptoms of Parkinson's disease—a discovery that could one day lead to new treatments for people who suffer from this devastating neu

November 28, 2010

SAN FRANCISCO, CA—Scientists at the Gladstone Institute of Neurological Disease (GIND) in San Francisco have discovered a new strategy to prevent memory deficits in a mouse model of Alzheimer's disease.

October 13, 2010

SAN FRANCISCO, CA—Alzheimer's disease is an extremely complicated disease. Several proteins seem to be involved in its cause and progression.

September 23, 2010

SAN FRANCISCO, CA—Scientists at the Gladstone Institute of Neurological Disease (GIND) have uncovered new approaches to reduce toxic proteins in Alzheimer's disease (AD) and other neurodegenerative diseases. The results might lead to new treatments for these diseases.

September 9, 2010

SAN FRANCISCO, CA—Amyloid beta (Αβ) proteins, widely thought to cause Alzheimer's disease, block the transport of vital cargoes inside brain cells.

July 21, 2010

SAN FRANCISCO, CA—The Gladstone Institutes and the international pharmaceutical company H. Lundbeck A/S have announced a collaborative research agreement to study and identify therapeutic candidates for neurological diseases.

July 7, 2010

SAN FRANCISCO, CA—Scientists at the Gladstone Institute of Neurological Disease (GIND) and Stanford University have shown how key circuits in the brain control movement.

December 3, 2009

SAN FRANCISCO, CA—Stimulating the growth of new neurons to replace those lost in Alzheimer's disease (AD) is an intriguing therapeutic possibility. But will the factors that cause AD allow the new neurons to thrive and function normally?

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