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News and Highlights

February 8, 2017
Katherine Pollard, PhD, Director, Gladstone Insitutes

Katherine Pollard, PhD, director of the convergence science initiative at the Gladstone Institutes, has been selected as a Chan Zuckerberg Investigator.

February 8, 2017

A trio of Gladstone scientists are studying the basic biology of the heart to guide the discovery of new therapies for conditions such as congenital heart disease and heart failure.

February 1, 2017

Gladstone scientists are mapping the connections and functions of brain cells to identify new drug targets to treat psychiatric disorders.

January 17, 2017
Dr. Sheng Ding

Anti-cancer drug prevented less weight gain and caused mice to burn more calories thanks to higher levels of metabolism-boosting brown fat.

January 12, 2017
Steven Finkbeiner with robotic microscope

Scientists discover an enzyme that that could be targeted to treat both Alzheimer’s disease and frontotemporal dementia.

December 26, 2016
Drs. Finkbeiner and Skibinski

New details learned about a key cellular protein could lead to treatments for neurodegenerative diseases, such as Parkinson’s, Huntington’s, Alzheimer’s, and amyotrophic lateral sclerosis (ALS). 

December 15, 2016
Drs. Srivastava and Ang

Research on a gene mutation that causes holes in the hearts of infants revealed insights into how the heart develops and how it stays healthy.

December 6, 2016
Drs. Srivastava, Conklin, Bruneau, Haldar

A new biopharmaceutical company, Tenaya Therapeutics Inc., will build on discoveries in cardiovascular disease research made at the Gladstone Institutes, concentrating on regenerative medicine and drug discovery for heart failure.

November 16, 2016
Drs. Sanjabi and Khan

The delayed antiviral immune response allows the virus to remain undetected in the vagina, which can increase the risk of fetal infection during pregnancy.

November 12, 2016
Drs. Mucke and Paz

Gladstone scientists discover how to control the cacophony of electrical activity in the brain, stopping seizures and reducing deficits related to Alzheimer’s disease.

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